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PUBLICATIONS

Honoring the remarkable achievements of scientists who have cited our products in their groundbreaking work. Your research and innovation make an invaluable contribution to advancing scientific discovery, and we're proud to be a part of your success. Together, with researchers like you, we're pushing the boundaries of what’s possible in science and technology!

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Publication List

Correlation analysis between gut microbiota characteristics and melasma

Liu, C., He, D., Yu, A., Deng, Y., Wang, L., & Song, Z. (2022). Correlation analysis between gut microbiota characteristics and melasma. Frontiers in Microbiology, 13. https://doi.org/10.3389/fmicb.2022.1051653

Overexpression of pumpkin CpVQ30 increased susceptibility of tobacco to powdery mildew

Li, S., & Wang, P. (2024). Overexpression of pumpkin CpVQ30 increased susceptibility of tobacco to powdery mildew. Physiological and Molecular Plant Pathology, 102365. https://doi.org/10.1016/j.pmpp.2024.102365

Genome-Wide Identification of Na + /H + Antiporter (NHX) Genes in Sugar Beet (Beta vulgaris L.) and Their Regulated Expression under Salt Stress

Wu, G., Wang, J., & Li, S. (2019). Genome-Wide Identification of Na+/H+ Antiporter (NHX) Genes in Sugar Beet (Beta vulgaris L.) and Their Regulated Expression under Salt Stress. Genes, 10(5), 401. https://doi.org/10.3390/genes10050401

Notoginsenoside R1 can inhibit the interaction between FGF1 and VEGFA to retard podocyte apoptosis

Li, C., Zhong, H., Ma, J., Liang, Z., Zhang, L., Liu, T., & Fan, W. (2023). Notoginsenoside R1 can inhibit the interaction between FGF1 and VEGFA to retard podocyte apoptosis. BMC Endocrine Disorders, 23(1). https://doi.org/10.1186/s12902-023-01402-6

Methyltransferase-like (METTL) homologues participate in Nicotiana benthamiana antiviral responses

Yue, J., Lu, Y., Sun, Z., Guo, Y., León, D. S., Pasin, F., & Zhao, M. (2023). Methyltransferase-like (METTL) homologues participate in Nicotiana benthamiana antiviral responses. Plant Signaling & Behavior, 18(1). https://doi.org/10.1080/15592324.2023.2214760

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